Jon E. Grant, JD, MD

Widower denies suicidal thoughts in hospital, but acts on them at home

DuPage County (IL) Circuit Court

A 77-year-old man was hospitalized after complaining of chest pain. He reported attempting suicide the night before by taking pills. His wife had died 5 months previously.

When the psychiatrist evaluated the patient the next day, the patient assured him that he was no longer suicidal, refused inpatient admission, but agreed to enter outpatient therapy. The patient repeated this intent to the hospital social worker.

The psychiatrist arranged visits by a home health care nurse. The patient was discharged after a 2-day stay, and the nurse visited the following day. The patient assured the nurse that he was not suicidal and called the psychiatrist to make an appointment for the next week. Two days later, the patient stabbed himself to death at home.

The estate claimed the psychiatrist should have kept the patient hospitalized. The psychiatrist claimed that involuntary admission was not possible because the patient was not dangerous to himself or others. The patient’s toxicology screen was negative except for his prescription drugs.

• The jury decided for the defense
  

Alcoholic promises to attend AA, but takes his life on Christmas Day

Davidson County (TN) Circuit Court

A 44-year-old man with a long history of alcohol abuse and failed rehabilitation was involuntarily admitted to a hospital after threatening suicide. His blood alcohol level was 0.393, and he had threatened suicide at the same facility 8 months before. A court order gave the hospital authority to involuntarily detain him until a hearing the following week.

The next day, the patient was transferred from the detoxification center to the psychiatric unit and evaluated by the psychiatrist. The patient disavowed suicidal thoughts, and the psychiatrist discharged the patient the following day (Christmas Eve, 48 hours after admission). The psychiatrist based this decision partially on the patient’s promise to enter inpatient alcohol treatment and attend an Alcoholics Anonymous meeting within 2 days.

On Christmas Day, the patient shot himself and died. His blood alcohol content at the time of death was 0.303.

The patient’s estate charged that the final discharge was negligent, the discharge instructions were inadequate, and the psychiatrist and hospital’s assessments were inaccurate.

The hospital argued that it deferred to the psychiatrist in the discharge decision. The psychiatrist argued that state law defined holding an individual without “immediate risk of substantial harm” as a felony.

• The jury decided in favor of the defendant psychiatrist. A directed verdict was granted for the hospital.
  

Plaintiff: Discharge led to hemiplegia

Broward County (FL) Circuit Court

Police took into custody a 27-year-old woman who had been wandering a public road, apparently under the influence of illegal substances. The officers transported her to a hospital, where the emergency room staff admitted her for psychiatric evaluation.

The psychiatrist determined that involuntary admission was not appropriate. When the patient refused the psychiatrist’s recommendation for voluntary admission, she was discharged.

The patient then went to her mother’s house, began drinking, and became combative. She started brandishing a rifle. The next day, the weapon discharged and a bullet lodged in her spine at the L2 vertebra. The patient is now hemiplegic and has no bladder or bowel control. She alleged that the hospital and psychiatrist were negligent in not admitting her.

• The hospital reached a $50,000 settlement before trial; the jury returned a $190,007 award, with 90% of fault apportioned to the plaintiff and 10% to the psychiatrist. After setoffs, the plaintiff’s net award was $80.
  

Dr. Grant’s observations

These cases illustrate suicide risk factors psychiatrists must consider even when a patient denies suicidal thoughts or intent. Suicide risk factors these patients showed include:

• recent discharge from psychiatric facilities¹
  
• recent suicide attempt with fairly high lethality potential (overdosing on pills)
  
• depressive turmoil and psychological isolation (recent loss of spouse)
  
• older widowed male^2-3
  
• history of dangerous behavior when intoxicated⁴
  
• possible “holiday effect.”⁵
  

These cases reflect one of psychiatry’s more troubling job requirements: assessing whether a patient is safe to discharge or should be admitted involuntarily. Such situations force us to balance the civil liberties of the mentally ill with our responsibility to care for those who lack insight into their illnesses. This tension often weighs heavily on psychiatrists⁶ and is, unfortunately, rather common. A study at one hospital found that approximately 8.5% of emergency department visits resulted in involuntary admission.⁷

As the verdicts in these cases suggest, the legal system recognizes that psychiatrists cannot predict suicide.⁸ Mistakes in clinical judgment are not the same as negligence, however, and failure to assess suicide risk or intervene appropriately for the level of risk may result in successful negligence claims.

 

Standards for emergency short-term hospitalization vary from state to state, so familiarize yourself with your state’s standards. Although one standard for involuntary admission is often imminent threat of harm to self, do not base the threat of danger only on a patient’s self-report. One study of patients who committed suicide while hospitalized found that 78% denied suicidal thoughts at their last communication.⁹ However, “locking up” suicidal patients to prevent a malpractice suit is equally inappropriate.

Assess suicide risk during a thoroughly documented psychiatric examination with particular attention to the patient’s history of suicidal behavior. Record details of the assessment in the patient’s chart (Table) at the time of evaluation, and document how these clinical factors influence your final decision.

Involuntary hospitalization provides the immediate benefit of supervision in a safe environment, and patients can gain short-term therapeutic benefits from inpatient treatment whether or not the admission was voluntary.¹⁰ Patients may eventually recognize admission was helpful, but their attitudes about the process often do not become more positive. To ease the stress of involuntary admission:

• acknowledge the patient’s disapproval
  
• tell the patient why he’s being hospitalized
  
• inform the patient about his or her legal rights.
  

Carry out this discussion with respect for the patient’s dignity and wishes.

Table

Documenting suicide risk assessment

Include in patient’s chart…	Examples…
Short-term factors	Current suicidal ideation/plan, lethality potential, current stressors (bereavement, illness, loss of job), recent discharge from a psychiatric facility, time of year (holiday effect, anniversaries)
Long-term factors	History of suicidal behavior/attempts, personality factors (agitation, hopelessness), gender, age, marital status, substance abuse history, psychiatric illness (depression, bipolar disorder, schizophrenia)
Appropriate psychiatric interventions based on the assessed degree of risk	Involuntary admission, intensive monitoring, outpatient visits, home healthcare nursing, residential placement, substance abuse treatment
Sources of information used	Medical records, patient self-report, family report, observation

Widower denies suicidal thoughts in hospital, but acts on them at home

DuPage County (IL) Circuit Court

A 77-year-old man was hospitalized after complaining of chest pain. He reported attempting suicide the night before by taking pills. His wife had died 5 months previously.

When the psychiatrist evaluated the patient the next day, the patient assured him that he was no longer suicidal, refused inpatient admission, but agreed to enter outpatient therapy. The patient repeated this intent to the hospital social worker.

The psychiatrist arranged visits by a home health care nurse. The patient was discharged after a 2-day stay, and the nurse visited the following day. The patient assured the nurse that he was not suicidal and called the psychiatrist to make an appointment for the next week. Two days later, the patient stabbed himself to death at home.

The estate claimed the psychiatrist should have kept the patient hospitalized. The psychiatrist claimed that involuntary admission was not possible because the patient was not dangerous to himself or others. The patient’s toxicology screen was negative except for his prescription drugs.

• The jury decided for the defense
  

Alcoholic promises to attend AA, but takes his life on Christmas Day

Davidson County (TN) Circuit Court

A 44-year-old man with a long history of alcohol abuse and failed rehabilitation was involuntarily admitted to a hospital after threatening suicide. His blood alcohol level was 0.393, and he had threatened suicide at the same facility 8 months before. A court order gave the hospital authority to involuntarily detain him until a hearing the following week.

The next day, the patient was transferred from the detoxification center to the psychiatric unit and evaluated by the psychiatrist. The patient disavowed suicidal thoughts, and the psychiatrist discharged the patient the following day (Christmas Eve, 48 hours after admission). The psychiatrist based this decision partially on the patient’s promise to enter inpatient alcohol treatment and attend an Alcoholics Anonymous meeting within 2 days.

On Christmas Day, the patient shot himself and died. His blood alcohol content at the time of death was 0.303.

The patient’s estate charged that the final discharge was negligent, the discharge instructions were inadequate, and the psychiatrist and hospital’s assessments were inaccurate.

The hospital argued that it deferred to the psychiatrist in the discharge decision. The psychiatrist argued that state law defined holding an individual without “immediate risk of substantial harm” as a felony.

• The jury decided in favor of the defendant psychiatrist. A directed verdict was granted for the hospital.
  

Plaintiff: Discharge led to hemiplegia

Broward County (FL) Circuit Court

Police took into custody a 27-year-old woman who had been wandering a public road, apparently under the influence of illegal substances. The officers transported her to a hospital, where the emergency room staff admitted her for psychiatric evaluation.

The psychiatrist determined that involuntary admission was not appropriate. When the patient refused the psychiatrist’s recommendation for voluntary admission, she was discharged.

The patient then went to her mother’s house, began drinking, and became combative. She started brandishing a rifle. The next day, the weapon discharged and a bullet lodged in her spine at the L2 vertebra. The patient is now hemiplegic and has no bladder or bowel control. She alleged that the hospital and psychiatrist were negligent in not admitting her.

• The hospital reached a $50,000 settlement before trial; the jury returned a $190,007 award, with 90% of fault apportioned to the plaintiff and 10% to the psychiatrist. After setoffs, the plaintiff’s net award was $80.
  

Dr. Grant’s observations

These cases illustrate suicide risk factors psychiatrists must consider even when a patient denies suicidal thoughts or intent. Suicide risk factors these patients showed include:

• recent discharge from psychiatric facilities¹
  
• recent suicide attempt with fairly high lethality potential (overdosing on pills)
  
• depressive turmoil and psychological isolation (recent loss of spouse)
  
• older widowed male^2-3
  
• history of dangerous behavior when intoxicated⁴
  
• possible “holiday effect.”⁵
  

These cases reflect one of psychiatry’s more troubling job requirements: assessing whether a patient is safe to discharge or should be admitted involuntarily. Such situations force us to balance the civil liberties of the mentally ill with our responsibility to care for those who lack insight into their illnesses. This tension often weighs heavily on psychiatrists⁶ and is, unfortunately, rather common. A study at one hospital found that approximately 8.5% of emergency department visits resulted in involuntary admission.⁷

As the verdicts in these cases suggest, the legal system recognizes that psychiatrists cannot predict suicide.⁸ Mistakes in clinical judgment are not the same as negligence, however, and failure to assess suicide risk or intervene appropriately for the level of risk may result in successful negligence claims.

 

Standards for emergency short-term hospitalization vary from state to state, so familiarize yourself with your state’s standards. Although one standard for involuntary admission is often imminent threat of harm to self, do not base the threat of danger only on a patient’s self-report. One study of patients who committed suicide while hospitalized found that 78% denied suicidal thoughts at their last communication.⁹ However, “locking up” suicidal patients to prevent a malpractice suit is equally inappropriate.

Assess suicide risk during a thoroughly documented psychiatric examination with particular attention to the patient’s history of suicidal behavior. Record details of the assessment in the patient’s chart (Table) at the time of evaluation, and document how these clinical factors influence your final decision.

Involuntary hospitalization provides the immediate benefit of supervision in a safe environment, and patients can gain short-term therapeutic benefits from inpatient treatment whether or not the admission was voluntary.¹⁰ Patients may eventually recognize admission was helpful, but their attitudes about the process often do not become more positive. To ease the stress of involuntary admission:

• acknowledge the patient’s disapproval
  
• tell the patient why he’s being hospitalized
  
• inform the patient about his or her legal rights.
  

Carry out this discussion with respect for the patient’s dignity and wishes.

Table

Documenting suicide risk assessment

Include in patient’s chart…	Examples…
Short-term factors	Current suicidal ideation/plan, lethality potential, current stressors (bereavement, illness, loss of job), recent discharge from a psychiatric facility, time of year (holiday effect, anniversaries)
Long-term factors	History of suicidal behavior/attempts, personality factors (agitation, hopelessness), gender, age, marital status, substance abuse history, psychiatric illness (depression, bipolar disorder, schizophrenia)
Appropriate psychiatric interventions based on the assessed degree of risk	Involuntary admission, intensive monitoring, outpatient visits, home healthcare nursing, residential placement, substance abuse treatment
Sources of information used	Medical records, patient self-report, family report, observation

Widower denies suicidal thoughts in hospital, but acts on them at home

DuPage County (IL) Circuit Court

A 77-year-old man was hospitalized after complaining of chest pain. He reported attempting suicide the night before by taking pills. His wife had died 5 months previously.

When the psychiatrist evaluated the patient the next day, the patient assured him that he was no longer suicidal, refused inpatient admission, but agreed to enter outpatient therapy. The patient repeated this intent to the hospital social worker.

The psychiatrist arranged visits by a home health care nurse. The patient was discharged after a 2-day stay, and the nurse visited the following day. The patient assured the nurse that he was not suicidal and called the psychiatrist to make an appointment for the next week. Two days later, the patient stabbed himself to death at home.

The estate claimed the psychiatrist should have kept the patient hospitalized. The psychiatrist claimed that involuntary admission was not possible because the patient was not dangerous to himself or others. The patient’s toxicology screen was negative except for his prescription drugs.

• The jury decided for the defense
  

Alcoholic promises to attend AA, but takes his life on Christmas Day

Davidson County (TN) Circuit Court

A 44-year-old man with a long history of alcohol abuse and failed rehabilitation was involuntarily admitted to a hospital after threatening suicide. His blood alcohol level was 0.393, and he had threatened suicide at the same facility 8 months before. A court order gave the hospital authority to involuntarily detain him until a hearing the following week.

The next day, the patient was transferred from the detoxification center to the psychiatric unit and evaluated by the psychiatrist. The patient disavowed suicidal thoughts, and the psychiatrist discharged the patient the following day (Christmas Eve, 48 hours after admission). The psychiatrist based this decision partially on the patient’s promise to enter inpatient alcohol treatment and attend an Alcoholics Anonymous meeting within 2 days.

On Christmas Day, the patient shot himself and died. His blood alcohol content at the time of death was 0.303.

The patient’s estate charged that the final discharge was negligent, the discharge instructions were inadequate, and the psychiatrist and hospital’s assessments were inaccurate.

The hospital argued that it deferred to the psychiatrist in the discharge decision. The psychiatrist argued that state law defined holding an individual without “immediate risk of substantial harm” as a felony.

• The jury decided in favor of the defendant psychiatrist. A directed verdict was granted for the hospital.
  

Plaintiff: Discharge led to hemiplegia

Broward County (FL) Circuit Court

Police took into custody a 27-year-old woman who had been wandering a public road, apparently under the influence of illegal substances. The officers transported her to a hospital, where the emergency room staff admitted her for psychiatric evaluation.

The psychiatrist determined that involuntary admission was not appropriate. When the patient refused the psychiatrist’s recommendation for voluntary admission, she was discharged.

The patient then went to her mother’s house, began drinking, and became combative. She started brandishing a rifle. The next day, the weapon discharged and a bullet lodged in her spine at the L2 vertebra. The patient is now hemiplegic and has no bladder or bowel control. She alleged that the hospital and psychiatrist were negligent in not admitting her.

• The hospital reached a $50,000 settlement before trial; the jury returned a $190,007 award, with 90% of fault apportioned to the plaintiff and 10% to the psychiatrist. After setoffs, the plaintiff’s net award was $80.
  

Dr. Grant’s observations

These cases illustrate suicide risk factors psychiatrists must consider even when a patient denies suicidal thoughts or intent. Suicide risk factors these patients showed include:

• recent discharge from psychiatric facilities¹
  
• recent suicide attempt with fairly high lethality potential (overdosing on pills)
  
• depressive turmoil and psychological isolation (recent loss of spouse)
  
• older widowed male^2-3
  
• history of dangerous behavior when intoxicated⁴
  
• possible “holiday effect.”⁵
  

These cases reflect one of psychiatry’s more troubling job requirements: assessing whether a patient is safe to discharge or should be admitted involuntarily. Such situations force us to balance the civil liberties of the mentally ill with our responsibility to care for those who lack insight into their illnesses. This tension often weighs heavily on psychiatrists⁶ and is, unfortunately, rather common. A study at one hospital found that approximately 8.5% of emergency department visits resulted in involuntary admission.⁷

As the verdicts in these cases suggest, the legal system recognizes that psychiatrists cannot predict suicide.⁸ Mistakes in clinical judgment are not the same as negligence, however, and failure to assess suicide risk or intervene appropriately for the level of risk may result in successful negligence claims.

 

Standards for emergency short-term hospitalization vary from state to state, so familiarize yourself with your state’s standards. Although one standard for involuntary admission is often imminent threat of harm to self, do not base the threat of danger only on a patient’s self-report. One study of patients who committed suicide while hospitalized found that 78% denied suicidal thoughts at their last communication.⁹ However, “locking up” suicidal patients to prevent a malpractice suit is equally inappropriate.

Assess suicide risk during a thoroughly documented psychiatric examination with particular attention to the patient’s history of suicidal behavior. Record details of the assessment in the patient’s chart (Table) at the time of evaluation, and document how these clinical factors influence your final decision.

Involuntary hospitalization provides the immediate benefit of supervision in a safe environment, and patients can gain short-term therapeutic benefits from inpatient treatment whether or not the admission was voluntary.¹⁰ Patients may eventually recognize admission was helpful, but their attitudes about the process often do not become more positive. To ease the stress of involuntary admission:

• acknowledge the patient’s disapproval
  
• tell the patient why he’s being hospitalized
  
• inform the patient about his or her legal rights.
  

Carry out this discussion with respect for the patient’s dignity and wishes.

Table

Documenting suicide risk assessment

Include in patient’s chart…	Examples…
Short-term factors	Current suicidal ideation/plan, lethality potential, current stressors (bereavement, illness, loss of job), recent discharge from a psychiatric facility, time of year (holiday effect, anniversaries)
Long-term factors	History of suicidal behavior/attempts, personality factors (agitation, hopelessness), gender, age, marital status, substance abuse history, psychiatric illness (depression, bipolar disorder, schizophrenia)
Appropriate psychiatric interventions based on the assessed degree of risk	Involuntary admission, intensive monitoring, outpatient visits, home healthcare nursing, residential placement, substance abuse treatment
Sources of information used	Medical records, patient self-report, family report, observation

Lying in the hospital bed, her face covered in bandages, Ms. S talked of suicide while awaiting reconstructive surgery on her nose: “If the only way to stop is by killing myself, I will.” When asked what she wanted to stop, she replied, “The picking. I pick at my face all day, every day.”

Ms. S, age 22, had picked a hole through the bridge of her nose, and her face was scarred and covered with scabs. Every morning for 5 years, she had gotten up, dressed, and then—after washing her face—felt intense, uncontrollable urges to pick at her face. Hours would go by and she was still picking, even as her face started to bleed: “I try to resist, but I can’t.”

Ms. S started picking her face when she was 17. She missed so much of high school because of time spent picking that she did not graduate. She now lives alone on medical disability. Conscious of her facial scarring, she rarely goes out in public. “People stare at me as if I’m a zoo animal; it’s so painful,” she says.

After her plastic surgery, she told the staff psychiatrist she had never sought help because she thought no one would understand her behavior. “It doesn’t make sense to me, and I’ve lived with it for years.”

Patients such as Ms. S often suffer in isolation for years, unaware that skin picking is a psychiatric disorder that can be treated successfully. Some are referred to psychiatrists through hospital emergency rooms or by dermatologists; others commit suicide, as Ms. S threatened to do.

In our practice, we recognize skin picking in patients with comorbid mood and anxiety disorders, body dysmorphic disorder (BDD), substance use disorders, impulse control disorders such as trichotillomania or kleptomania, and personality disorders.

Based on recent evidence and our experience, we discuss three steps to help you diagnose pathologic skin picking. We then examine treatment options that have shown benefit for skinpicking patients, including habit reversal psychotherapy and medications.

WHAT IS SKIN PICKING?

Pathologic skin picking is repetitive, ritualistic, or impulsive picking of normal skin, leading to tissue damage, personal distress, and impaired functioning.¹ The behavior has been described for more than 100 years but remains poorly understood, under-diagnosed, and under-treated.^2,3

Most people pick at their hands or face to a limited extent,⁴ and picking does not by itself suggest a psychiatric disorder. Pathology exists in the focus, duration, and extent of the behavior, as well as reasons for picking, associated emotions, and resulting problems. Persons with pathologic skin picking report irresistible, intrusive, and/or senseless thoughts of picking or impulses to pick, accompanied by marked distress.¹ Pathologic skin picking is recurrent and usually results in noticeable skin damage, although many patients try to camouflage the lesions or scarring with makeup.

Pathologic skin picking’s prevalence is unknown. One early study estimated that 2% of dermatology patients suffer from skin picking.¹ Two clinical studies found that 3.8% of college students⁴ and 28% of patients with BDD meet diagnostic criteria.^5,6

TWEEZERS, RAZOR BLADES, KNIVES…

Persons who engage in pathologic skin picking typically spend substantial time picking. Most often they pick the face, but any body part—lips, arms, hands, or legs—may be the focus. They may pick at blemishes, pimples, scars, or healthy skin. Some use their hands and fingernails to pick, and others use pins, tweezers, razor blades, or knives. Picking may worsen in the evening.^2,7

Although picking episodes may last only a few minutes, many patients have multiple episodes each day. Some pick for as long as 12 hours every day,^2,5 which often leads to scarring and disfigurement. In one study, 90% of patients had at least minor tissue damage, 61% suffered infections, and 45% had “deep craters” because of picking.²

Reasons for picking. Many patients pick to relieve discomfort or tension.¹ Others pick to improve their appearance, as in BDD, or to remove perceived dirt or contaminants, as in obsessive-compulsive disorder (OCD).^1,5 Still others say they pick as a habit, with minimal awareness.¹ Itching or uneven skin may also cause the behavior.^1,3 We have found that a patient may pick for several of these reasons. Most report:

• tension before picking
• satisfaction during picking
• guilt, shame, and dysphoria after picking.¹

Social impairment. Shame after picking episodes often leads patients to cover lesions with clothing or makeup and to avoid social contact.⁷ Substantial social and occupational impairment have been reported^3,5,7 because of the hours spent picking and from avoiding people because of disfigurement.

Physical injury. Skin picking may cause serious injuries. Some of our patients have required emergency medical intervention and sutures after picking through a major blood vessel (such as the facial artery). One woman—who picked at a pimple on her neck with tweezers—lacerated her carotid artery, causing a near-fatal hemorrhage that required emergency surgery.⁸

 

Suicide risk. In a series of 123 patients with BDD, 33 (27%) excessively picked their skin and 10 of those who picked their skin (33%) had attempted suicide.5 In a case series of 31 patients with skin picking, 10% had attempted suicide.² We know of several young women whose chief complaint was skin picking and who committed suicide.⁵

Gender. The gender ratio of patients with skin picking remains unclear. In two case series that totaled 65 patients, 87% to 92% of those with pathologic skin picking were female.^2,7 In the series of patients with BDD, 58% of the 33 who compulsively picked their skin were female.⁵ On the other hand, most of 28 patients seen in a dermatology clinic for neurotic excoriations were male.⁹

Onset and chronicity. Pathologic skin picking may develop at any age, but it usually manifests in late adolescence or early adulthood, often after onset of a dermatologic illness such as acne² or in response to itching.³ Although long-term studies have not been done, the disorder appears to often be chronic, with waxing and waning of picking intensity and frequency.^1,2

Table 1

Skin picking: 3 steps to diagnosis and treatment

Step 1: Assess reasons for skin picking  Dermatologic or medical disorder? atopic dermatitis scabies Prader-Willi syndrome  Psychiatric disorder? body dysmorphic disorder obsessive-compulsive disorder delusional disorder dermatitis artefacta  Impulse control disorder, not otherwise specified?
Step 2: Assess picking severity  Treat comorbid mood or anxiety disorders  Treat skin picking if: patient is preoccupied with picking picking causes distress or dysfunction picking is causing skin lesions/disfigurement
Step 3: Provide recommended treatment  For adults  Habit reversal therapy plus medication is usually necessary  For children and adolescents  Habit reversal therapy alone for mild to moderate symptoms  Habit reversal therapy plus medication for severe symptoms

Comorbid psychopathology. In clinical settings, common comorbid psychopathologies include mood disorders (in 48% to 68% of patients with skin picking), anxiety disorders (41% to 64%), and alcohol use disorders (39%).²

In one patient sample, 71% of skin pickers met criteria for at least one personality disorder (48% had obsessive-compulsive personality disorder, and 26% met criteria for borderline personality disorder).²

Table 2

Medications with evidence of benefit for skin picking*

Medication	Dosage	Type of evidence
SSRIs
Citalopram	40 mg/d	Case report (effective only with inositol augmentation)16
Fluoxetine	20 to 80 mg/d	Case reports5,14-15 and two double-blind studies23-24
Fluvoxamine	100 to 300 mg/d	Case report,8 open-label study,21 and double-blind trial22
Sertraline	50 to 200 mg/d	Open-label study9
Other agents
Clomipramine	50 mg/d	Case report3
Doxepin	30 mg/d	Case report1
Naltrexone	50 mg/d	Case report20
Olanzapine	2.5 to 7.5 mg/d	Case report17
Pimozide	4 mg/d	Case report18
* Off-label uses; little scientific evidence supports using medications other than SSRIs for treating skin picking. Inform patients of the evidence for using any medication, risk of side effects including change in cardiac conduction (pimozide, clomipramine), seizure risk (pimozide, clomipramine), and tardive dyskinesia (pimozide), and potential interactions with other medications (all of the above).

PRIMARY VS. SECONDARY DISORDER

Is skin picking an independent disorder or a symptom of other psychiatric disorders? Although skin picking is not included in DSM-IV and has no formal diagnostic criteria, some forms of this behavior may belong among the impulse control disorders.

Patients often report an urge to pick their skin in response to increasing tension,^1,3 and picking results in transient relief or pleasure.^1,2 This description mirrors that of other impulse control disorders, such as trichotillomania and kleptomania. In fact, one study found that trichotillomania and kleptomania were common comorbidities among patients with skin picking (23% and 16%, respectively).² In 34 patients with psychogenic excoriation, only 7 (21%) appeared to have skin picking as a primary complaint, unaccounted for by another psychiatric disorder.⁷

Skin picking may also be a symptom of other psychiatric disorders. To determine whether another disorder is present, we ask patients why they pick their skin. Patients may be reluctant to reveal either the picking or the underlying disorder because of embarrassment and shame. The diagnosis can often be clarified by asking about the following conditions:

Body dysmorphic disorder. Nearly 30% of patients with BDD pick their skin to a pathologic extent.^5,6 The purpose of picking in BDD is to remove or minimize a nonexistent or slight imperfection in appearance (such as scars, pimples, bumps).^5,6

Obsessive-compulsive disorder. Patients with OCD may pick their skin in response to contamination obsessions.¹ Picking is often repetitive and ritualistic, and—as with compulsions—the behavior may reduce tension.¹⁰

Genetic disorders. Skin picking may be a symptom of Prader-Willi syndrome, a genetic disorder characterized by muscular hypotonia, short stature, characteristic facial features, intellectual disabilities, hypogonadism, hyperphagia, and an increased obesity risk. In one study, 97% of patients with Prader-Willi syndrome engaged in skin picking.¹¹

 

Delusional disorder. Delusions of parasitosis may result in skin picking, as patients attempt to remove imagined parasites or other vermin from on or under their skin.¹²

Dermatitis artefacta. Patients may consciously create skin lesions to assume the sick role. Onethird of patients presenting to dermatologists with a disease that is primarily psychiatric may be suffering from dermatitis artefacta.¹³

TREATMENT RECOMMENDATIONS

Successful clinical care of pathologic skin picking requires perseverance and patience from both patient and clinician.

Treatment begins with a thorough dermatologic examination for medical causes of skin picking (such as atopic dermatitis or scabies) and to treat excoriations (such as with antibiotics for infection). After the dermatologist has ruled out a medical cause, carefully assess the patient’s picking behavior and related psychiatric problems (Table 1).

• If picking is secondary to a psychiatric disorder, begin by providing appropriate treatment for that disorder.
• If picking results from BDD or OCD, we recommend habit reversal therapy combined with medication.
• If picking appears to be an independent impulse control disorder, simultaneous habit reversal therapy and medication is usually necessary to reduce symptoms.

SSRIs are a reasonable first medication because of evidence for their efficacy in reducing skin picking. Higher dosages—comparable to those used in treating OCD—are usually required to improve skin-picking behavior. You may need to try another SSRI if the first trial results in partial or no response.

In our experience, augmenting an SSRI with naltrexone, 50 mg/d, helps reduce intrusive urges to pick and is worth considering if SSRI therapy results in only partial response.

Children or adolescents. Depending upon symptom severity, a trial of habit reversal therapy may be appropriate before you recommend using medication.

EVIDENCE FOR DRUG THERAPY

Although few treatment studies have been done, skin picking does appear to respond to medication (Table 2).

Because no medications are approved to treat skin-picking behavior, inform patients of any “off-label” uses and the scientific or clinical evidence for considering medication treatment.

Case reports and case series. Selective serotonin reuptake inhibitors (SSRIs) appear most effective in patients with picking behavior, including:

• fluvoxamine, 300 mg/d, in one case report⁸
• fluoxetine, 20 to 80 mg/d, in several case reports.^5,14-15

In a series of 33 patients with BDD and compulsive skin picking, one-half (49%) of a variety of SSRI treatment trials improved BDD symptoms and skin picking behavior. The percentage of patients who improved was not examined. Dermatologic treatment alone was effective for only 15% of patients.⁵

Medications other than SSRIs have also been studied. One patient improved within 3 weeks of taking the tricyclic antidepressant clomipramine, 50 mg/d.³ Another patient picked her skin less often 4 weeks after inositol, 18 grams/d, was added to citalopram, 40 mg/d. Inositol, a nonprescription isomer of glucose, is a precursor in the phosphatidylinositol second-messenger cycle, which may play a role at certain serotonin receptors.¹⁶ The patient was given 6 grams dissolved in water three times daily.

Case reports have also suggested that olanzapine, pimozide, doxepin, and naltrexone may be beneficial in reducing skin excoriations. These reports often involved patients with psychiatric and medical comorbidities.^17-20

Table 3

Habit reversal: 5 components in patient learning

Awareness about picking behavior
Relaxation to reduce anxiety
Competing responses to learn behaviors incompatible with picking (such as fist clenching)
Rewarding oneself for successfully resisting picking
Generalizing the behavioral control

Open-label studies. In an open-label study of 28 patients with neurotic excoriation treated in a dermatology clinic, 68% improved within 1 month with sertraline, mean dosage 95 mg/d.⁹ Similarly, open-label fluvoxamine, mean dosage 112.5 mg/d, was effective in reducing skin excoriation in 7 of 14 patients treated for 12 weeks in a psychiatric setting.²¹

Double-blind studies. In a double-blind study using fluvoxamine with supportive psychotherapy in patients with psychocutaneous disorders, all five patients with acne excoriee improved after 4 weeks of medication treatment (none was randomized to placebo).²²

In a 10-week, double-blind study, 10 patients were assigned to fluoxetine, mean dosage 53.0 ± 16.4 mg/d, and 11 to placebo. A patient self-report visual analog scale showed that fluoxetine was significantly more effective than placebo in reducing picking behavior. Two other measures did not show significant improvement, however, perhaps because of the small sample size.²³

In a third study, 8 of 15 patients responded to open-label fluoxetine, 20 to 60 mg/d after 6 weeks. The responders were then randomized to 6 additional weeks of fluoxetine or placebo. All four patients assigned to continue active medication maintained their improvement. Symptoms returned to baseline by week 12 in the four assigned to placebo.²⁴

EVIDENCE FOR HABIT REVERSAL THERAPY

No controlled trials have examined psychosocial treatments for skin picking, but several psychotherapeutic interventions appear promising. Habit reversal has shown promise in three case reports totaling seven patients and appears to reduce picking behavior within a few weeks.^25-27

 

In a case series, three patients were successfully treated with habit reversal (Table 3) and cognitive-behavioral techniques, consisting of:

• awareness training (using a skin-picking diary)
• competing response techniques (such as making a fist or squeezing a ball)
• emotion regulation skills
• psychoeducation
• cognitive restructuring (such as using Socratic questioning to produce rational alternatives) in situations that elicit the urge to pick.²⁸

In another case series, 22 dermatology patients with skin picking received psychotherapy with insight-oriented and behavioral components. Therapy included attention to developmental issues and active conflicts, cognitive restructuring, and tools to manage aggression and social relations. Although treatment duration varied— the mean was weekly for 14 months—skin lesions healed in 17 patients (77%).²⁹

Related resources

• Obsessive-Compulsive Foundation http://www.ocfoundation.org
• Koran LM. Obsessive-compulsive and related disorders in adults: A comprehensive clinical guide. Cambridge, UK: Cambridge University Press, 1999.
• Phillips KA. The broken mirror: Recognizing and treating body dysmorphic disorder. New York: Oxford University Press, 1996.

Drug brand names

• Citalopram • Celexa
• Clomipramine • Anafranil
• Doxepin • Sinequan
• Fluoxetine • Prozac
• Fluvoxamine • Luvox
• Naltrexone • ReVia
• Olanzapine • Zyprexa
• Pimozide • Orap
• Sertraline • Zoloft

Disclosure

Dr. Grant reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.

Dr. Phillips receives research support from Eli Lilly and Co., Forest Pharmaceuticals, and Gate Pharmaceuticals; she is a speaker for or consultant to Eli Lilly and Co., Forest Pharmaceuticals, and UCB Pharma.

Lying in the hospital bed, her face covered in bandages, Ms. S talked of suicide while awaiting reconstructive surgery on her nose: “If the only way to stop is by killing myself, I will.” When asked what she wanted to stop, she replied, “The picking. I pick at my face all day, every day.”

Ms. S, age 22, had picked a hole through the bridge of her nose, and her face was scarred and covered with scabs. Every morning for 5 years, she had gotten up, dressed, and then—after washing her face—felt intense, uncontrollable urges to pick at her face. Hours would go by and she was still picking, even as her face started to bleed: “I try to resist, but I can’t.”

Ms. S started picking her face when she was 17. She missed so much of high school because of time spent picking that she did not graduate. She now lives alone on medical disability. Conscious of her facial scarring, she rarely goes out in public. “People stare at me as if I’m a zoo animal; it’s so painful,” she says.

After her plastic surgery, she told the staff psychiatrist she had never sought help because she thought no one would understand her behavior. “It doesn’t make sense to me, and I’ve lived with it for years.”

Patients such as Ms. S often suffer in isolation for years, unaware that skin picking is a psychiatric disorder that can be treated successfully. Some are referred to psychiatrists through hospital emergency rooms or by dermatologists; others commit suicide, as Ms. S threatened to do.

In our practice, we recognize skin picking in patients with comorbid mood and anxiety disorders, body dysmorphic disorder (BDD), substance use disorders, impulse control disorders such as trichotillomania or kleptomania, and personality disorders.

Based on recent evidence and our experience, we discuss three steps to help you diagnose pathologic skin picking. We then examine treatment options that have shown benefit for skinpicking patients, including habit reversal psychotherapy and medications.

WHAT IS SKIN PICKING?

Pathologic skin picking is repetitive, ritualistic, or impulsive picking of normal skin, leading to tissue damage, personal distress, and impaired functioning.¹ The behavior has been described for more than 100 years but remains poorly understood, under-diagnosed, and under-treated.^2,3

Most people pick at their hands or face to a limited extent,⁴ and picking does not by itself suggest a psychiatric disorder. Pathology exists in the focus, duration, and extent of the behavior, as well as reasons for picking, associated emotions, and resulting problems. Persons with pathologic skin picking report irresistible, intrusive, and/or senseless thoughts of picking or impulses to pick, accompanied by marked distress.¹ Pathologic skin picking is recurrent and usually results in noticeable skin damage, although many patients try to camouflage the lesions or scarring with makeup.

Pathologic skin picking’s prevalence is unknown. One early study estimated that 2% of dermatology patients suffer from skin picking.¹ Two clinical studies found that 3.8% of college students⁴ and 28% of patients with BDD meet diagnostic criteria.^5,6

TWEEZERS, RAZOR BLADES, KNIVES…

Persons who engage in pathologic skin picking typically spend substantial time picking. Most often they pick the face, but any body part—lips, arms, hands, or legs—may be the focus. They may pick at blemishes, pimples, scars, or healthy skin. Some use their hands and fingernails to pick, and others use pins, tweezers, razor blades, or knives. Picking may worsen in the evening.^2,7

Although picking episodes may last only a few minutes, many patients have multiple episodes each day. Some pick for as long as 12 hours every day,^2,5 which often leads to scarring and disfigurement. In one study, 90% of patients had at least minor tissue damage, 61% suffered infections, and 45% had “deep craters” because of picking.²

Reasons for picking. Many patients pick to relieve discomfort or tension.¹ Others pick to improve their appearance, as in BDD, or to remove perceived dirt or contaminants, as in obsessive-compulsive disorder (OCD).^1,5 Still others say they pick as a habit, with minimal awareness.¹ Itching or uneven skin may also cause the behavior.^1,3 We have found that a patient may pick for several of these reasons. Most report:

• tension before picking
• satisfaction during picking
• guilt, shame, and dysphoria after picking.¹

Social impairment. Shame after picking episodes often leads patients to cover lesions with clothing or makeup and to avoid social contact.⁷ Substantial social and occupational impairment have been reported^3,5,7 because of the hours spent picking and from avoiding people because of disfigurement.

Physical injury. Skin picking may cause serious injuries. Some of our patients have required emergency medical intervention and sutures after picking through a major blood vessel (such as the facial artery). One woman—who picked at a pimple on her neck with tweezers—lacerated her carotid artery, causing a near-fatal hemorrhage that required emergency surgery.⁸

 

Suicide risk. In a series of 123 patients with BDD, 33 (27%) excessively picked their skin and 10 of those who picked their skin (33%) had attempted suicide.5 In a case series of 31 patients with skin picking, 10% had attempted suicide.² We know of several young women whose chief complaint was skin picking and who committed suicide.⁵

Gender. The gender ratio of patients with skin picking remains unclear. In two case series that totaled 65 patients, 87% to 92% of those with pathologic skin picking were female.^2,7 In the series of patients with BDD, 58% of the 33 who compulsively picked their skin were female.⁵ On the other hand, most of 28 patients seen in a dermatology clinic for neurotic excoriations were male.⁹

Onset and chronicity. Pathologic skin picking may develop at any age, but it usually manifests in late adolescence or early adulthood, often after onset of a dermatologic illness such as acne² or in response to itching.³ Although long-term studies have not been done, the disorder appears to often be chronic, with waxing and waning of picking intensity and frequency.^1,2

Table 1

Skin picking: 3 steps to diagnosis and treatment

Step 1: Assess reasons for skin picking  Dermatologic or medical disorder? atopic dermatitis scabies Prader-Willi syndrome  Psychiatric disorder? body dysmorphic disorder obsessive-compulsive disorder delusional disorder dermatitis artefacta  Impulse control disorder, not otherwise specified?
Step 2: Assess picking severity  Treat comorbid mood or anxiety disorders  Treat skin picking if: patient is preoccupied with picking picking causes distress or dysfunction picking is causing skin lesions/disfigurement
Step 3: Provide recommended treatment  For adults  Habit reversal therapy plus medication is usually necessary  For children and adolescents  Habit reversal therapy alone for mild to moderate symptoms  Habit reversal therapy plus medication for severe symptoms

Comorbid psychopathology. In clinical settings, common comorbid psychopathologies include mood disorders (in 48% to 68% of patients with skin picking), anxiety disorders (41% to 64%), and alcohol use disorders (39%).²

In one patient sample, 71% of skin pickers met criteria for at least one personality disorder (48% had obsessive-compulsive personality disorder, and 26% met criteria for borderline personality disorder).²

Table 2

Medications with evidence of benefit for skin picking*

Medication	Dosage	Type of evidence
SSRIs
Citalopram	40 mg/d	Case report (effective only with inositol augmentation)16
Fluoxetine	20 to 80 mg/d	Case reports5,14-15 and two double-blind studies23-24
Fluvoxamine	100 to 300 mg/d	Case report,8 open-label study,21 and double-blind trial22
Sertraline	50 to 200 mg/d	Open-label study9
Other agents
Clomipramine	50 mg/d	Case report3
Doxepin	30 mg/d	Case report1
Naltrexone	50 mg/d	Case report20
Olanzapine	2.5 to 7.5 mg/d	Case report17
Pimozide	4 mg/d	Case report18
* Off-label uses; little scientific evidence supports using medications other than SSRIs for treating skin picking. Inform patients of the evidence for using any medication, risk of side effects including change in cardiac conduction (pimozide, clomipramine), seizure risk (pimozide, clomipramine), and tardive dyskinesia (pimozide), and potential interactions with other medications (all of the above).

PRIMARY VS. SECONDARY DISORDER

Is skin picking an independent disorder or a symptom of other psychiatric disorders? Although skin picking is not included in DSM-IV and has no formal diagnostic criteria, some forms of this behavior may belong among the impulse control disorders.

Patients often report an urge to pick their skin in response to increasing tension,^1,3 and picking results in transient relief or pleasure.^1,2 This description mirrors that of other impulse control disorders, such as trichotillomania and kleptomania. In fact, one study found that trichotillomania and kleptomania were common comorbidities among patients with skin picking (23% and 16%, respectively).² In 34 patients with psychogenic excoriation, only 7 (21%) appeared to have skin picking as a primary complaint, unaccounted for by another psychiatric disorder.⁷

Skin picking may also be a symptom of other psychiatric disorders. To determine whether another disorder is present, we ask patients why they pick their skin. Patients may be reluctant to reveal either the picking or the underlying disorder because of embarrassment and shame. The diagnosis can often be clarified by asking about the following conditions:

Body dysmorphic disorder. Nearly 30% of patients with BDD pick their skin to a pathologic extent.^5,6 The purpose of picking in BDD is to remove or minimize a nonexistent or slight imperfection in appearance (such as scars, pimples, bumps).^5,6

Obsessive-compulsive disorder. Patients with OCD may pick their skin in response to contamination obsessions.¹ Picking is often repetitive and ritualistic, and—as with compulsions—the behavior may reduce tension.¹⁰

Genetic disorders. Skin picking may be a symptom of Prader-Willi syndrome, a genetic disorder characterized by muscular hypotonia, short stature, characteristic facial features, intellectual disabilities, hypogonadism, hyperphagia, and an increased obesity risk. In one study, 97% of patients with Prader-Willi syndrome engaged in skin picking.¹¹

 

Delusional disorder. Delusions of parasitosis may result in skin picking, as patients attempt to remove imagined parasites or other vermin from on or under their skin.¹²

Dermatitis artefacta. Patients may consciously create skin lesions to assume the sick role. Onethird of patients presenting to dermatologists with a disease that is primarily psychiatric may be suffering from dermatitis artefacta.¹³

TREATMENT RECOMMENDATIONS

Successful clinical care of pathologic skin picking requires perseverance and patience from both patient and clinician.

Treatment begins with a thorough dermatologic examination for medical causes of skin picking (such as atopic dermatitis or scabies) and to treat excoriations (such as with antibiotics for infection). After the dermatologist has ruled out a medical cause, carefully assess the patient’s picking behavior and related psychiatric problems (Table 1).

• If picking is secondary to a psychiatric disorder, begin by providing appropriate treatment for that disorder.
• If picking results from BDD or OCD, we recommend habit reversal therapy combined with medication.
• If picking appears to be an independent impulse control disorder, simultaneous habit reversal therapy and medication is usually necessary to reduce symptoms.

SSRIs are a reasonable first medication because of evidence for their efficacy in reducing skin picking. Higher dosages—comparable to those used in treating OCD—are usually required to improve skin-picking behavior. You may need to try another SSRI if the first trial results in partial or no response.

In our experience, augmenting an SSRI with naltrexone, 50 mg/d, helps reduce intrusive urges to pick and is worth considering if SSRI therapy results in only partial response.

Children or adolescents. Depending upon symptom severity, a trial of habit reversal therapy may be appropriate before you recommend using medication.

EVIDENCE FOR DRUG THERAPY

Although few treatment studies have been done, skin picking does appear to respond to medication (Table 2).

Because no medications are approved to treat skin-picking behavior, inform patients of any “off-label” uses and the scientific or clinical evidence for considering medication treatment.

Case reports and case series. Selective serotonin reuptake inhibitors (SSRIs) appear most effective in patients with picking behavior, including:

• fluvoxamine, 300 mg/d, in one case report⁸
• fluoxetine, 20 to 80 mg/d, in several case reports.^5,14-15

In a series of 33 patients with BDD and compulsive skin picking, one-half (49%) of a variety of SSRI treatment trials improved BDD symptoms and skin picking behavior. The percentage of patients who improved was not examined. Dermatologic treatment alone was effective for only 15% of patients.⁵

Medications other than SSRIs have also been studied. One patient improved within 3 weeks of taking the tricyclic antidepressant clomipramine, 50 mg/d.³ Another patient picked her skin less often 4 weeks after inositol, 18 grams/d, was added to citalopram, 40 mg/d. Inositol, a nonprescription isomer of glucose, is a precursor in the phosphatidylinositol second-messenger cycle, which may play a role at certain serotonin receptors.¹⁶ The patient was given 6 grams dissolved in water three times daily.

Case reports have also suggested that olanzapine, pimozide, doxepin, and naltrexone may be beneficial in reducing skin excoriations. These reports often involved patients with psychiatric and medical comorbidities.^17-20

Table 3

Habit reversal: 5 components in patient learning

Awareness about picking behavior
Relaxation to reduce anxiety
Competing responses to learn behaviors incompatible with picking (such as fist clenching)
Rewarding oneself for successfully resisting picking
Generalizing the behavioral control

Open-label studies. In an open-label study of 28 patients with neurotic excoriation treated in a dermatology clinic, 68% improved within 1 month with sertraline, mean dosage 95 mg/d.⁹ Similarly, open-label fluvoxamine, mean dosage 112.5 mg/d, was effective in reducing skin excoriation in 7 of 14 patients treated for 12 weeks in a psychiatric setting.²¹

Double-blind studies. In a double-blind study using fluvoxamine with supportive psychotherapy in patients with psychocutaneous disorders, all five patients with acne excoriee improved after 4 weeks of medication treatment (none was randomized to placebo).²²

In a 10-week, double-blind study, 10 patients were assigned to fluoxetine, mean dosage 53.0 ± 16.4 mg/d, and 11 to placebo. A patient self-report visual analog scale showed that fluoxetine was significantly more effective than placebo in reducing picking behavior. Two other measures did not show significant improvement, however, perhaps because of the small sample size.²³

In a third study, 8 of 15 patients responded to open-label fluoxetine, 20 to 60 mg/d after 6 weeks. The responders were then randomized to 6 additional weeks of fluoxetine or placebo. All four patients assigned to continue active medication maintained their improvement. Symptoms returned to baseline by week 12 in the four assigned to placebo.²⁴

EVIDENCE FOR HABIT REVERSAL THERAPY

No controlled trials have examined psychosocial treatments for skin picking, but several psychotherapeutic interventions appear promising. Habit reversal has shown promise in three case reports totaling seven patients and appears to reduce picking behavior within a few weeks.^25-27

 

In a case series, three patients were successfully treated with habit reversal (Table 3) and cognitive-behavioral techniques, consisting of:

• awareness training (using a skin-picking diary)
• competing response techniques (such as making a fist or squeezing a ball)
• emotion regulation skills
• psychoeducation
• cognitive restructuring (such as using Socratic questioning to produce rational alternatives) in situations that elicit the urge to pick.²⁸

In another case series, 22 dermatology patients with skin picking received psychotherapy with insight-oriented and behavioral components. Therapy included attention to developmental issues and active conflicts, cognitive restructuring, and tools to manage aggression and social relations. Although treatment duration varied— the mean was weekly for 14 months—skin lesions healed in 17 patients (77%).²⁹

Related resources

• Obsessive-Compulsive Foundation http://www.ocfoundation.org
• Koran LM. Obsessive-compulsive and related disorders in adults: A comprehensive clinical guide. Cambridge, UK: Cambridge University Press, 1999.
• Phillips KA. The broken mirror: Recognizing and treating body dysmorphic disorder. New York: Oxford University Press, 1996.

Drug brand names

• Citalopram • Celexa
• Clomipramine • Anafranil
• Doxepin • Sinequan
• Fluoxetine • Prozac
• Fluvoxamine • Luvox
• Naltrexone • ReVia
• Olanzapine • Zyprexa
• Pimozide • Orap
• Sertraline • Zoloft

Disclosure

Dr. Grant reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.

Dr. Phillips receives research support from Eli Lilly and Co., Forest Pharmaceuticals, and Gate Pharmaceuticals; she is a speaker for or consultant to Eli Lilly and Co., Forest Pharmaceuticals, and UCB Pharma.

Lying in the hospital bed, her face covered in bandages, Ms. S talked of suicide while awaiting reconstructive surgery on her nose: “If the only way to stop is by killing myself, I will.” When asked what she wanted to stop, she replied, “The picking. I pick at my face all day, every day.”

Ms. S, age 22, had picked a hole through the bridge of her nose, and her face was scarred and covered with scabs. Every morning for 5 years, she had gotten up, dressed, and then—after washing her face—felt intense, uncontrollable urges to pick at her face. Hours would go by and she was still picking, even as her face started to bleed: “I try to resist, but I can’t.”

Ms. S started picking her face when she was 17. She missed so much of high school because of time spent picking that she did not graduate. She now lives alone on medical disability. Conscious of her facial scarring, she rarely goes out in public. “People stare at me as if I’m a zoo animal; it’s so painful,” she says.

After her plastic surgery, she told the staff psychiatrist she had never sought help because she thought no one would understand her behavior. “It doesn’t make sense to me, and I’ve lived with it for years.”

Patients such as Ms. S often suffer in isolation for years, unaware that skin picking is a psychiatric disorder that can be treated successfully. Some are referred to psychiatrists through hospital emergency rooms or by dermatologists; others commit suicide, as Ms. S threatened to do.

In our practice, we recognize skin picking in patients with comorbid mood and anxiety disorders, body dysmorphic disorder (BDD), substance use disorders, impulse control disorders such as trichotillomania or kleptomania, and personality disorders.

Based on recent evidence and our experience, we discuss three steps to help you diagnose pathologic skin picking. We then examine treatment options that have shown benefit for skinpicking patients, including habit reversal psychotherapy and medications.

WHAT IS SKIN PICKING?

Pathologic skin picking is repetitive, ritualistic, or impulsive picking of normal skin, leading to tissue damage, personal distress, and impaired functioning.¹ The behavior has been described for more than 100 years but remains poorly understood, under-diagnosed, and under-treated.^2,3

Most people pick at their hands or face to a limited extent,⁴ and picking does not by itself suggest a psychiatric disorder. Pathology exists in the focus, duration, and extent of the behavior, as well as reasons for picking, associated emotions, and resulting problems. Persons with pathologic skin picking report irresistible, intrusive, and/or senseless thoughts of picking or impulses to pick, accompanied by marked distress.¹ Pathologic skin picking is recurrent and usually results in noticeable skin damage, although many patients try to camouflage the lesions or scarring with makeup.

Pathologic skin picking’s prevalence is unknown. One early study estimated that 2% of dermatology patients suffer from skin picking.¹ Two clinical studies found that 3.8% of college students⁴ and 28% of patients with BDD meet diagnostic criteria.^5,6

TWEEZERS, RAZOR BLADES, KNIVES…

Persons who engage in pathologic skin picking typically spend substantial time picking. Most often they pick the face, but any body part—lips, arms, hands, or legs—may be the focus. They may pick at blemishes, pimples, scars, or healthy skin. Some use their hands and fingernails to pick, and others use pins, tweezers, razor blades, or knives. Picking may worsen in the evening.^2,7

Although picking episodes may last only a few minutes, many patients have multiple episodes each day. Some pick for as long as 12 hours every day,^2,5 which often leads to scarring and disfigurement. In one study, 90% of patients had at least minor tissue damage, 61% suffered infections, and 45% had “deep craters” because of picking.²

Reasons for picking. Many patients pick to relieve discomfort or tension.¹ Others pick to improve their appearance, as in BDD, or to remove perceived dirt or contaminants, as in obsessive-compulsive disorder (OCD).^1,5 Still others say they pick as a habit, with minimal awareness.¹ Itching or uneven skin may also cause the behavior.^1,3 We have found that a patient may pick for several of these reasons. Most report:

• tension before picking
• satisfaction during picking
• guilt, shame, and dysphoria after picking.¹

Social impairment. Shame after picking episodes often leads patients to cover lesions with clothing or makeup and to avoid social contact.⁷ Substantial social and occupational impairment have been reported^3,5,7 because of the hours spent picking and from avoiding people because of disfigurement.

Physical injury. Skin picking may cause serious injuries. Some of our patients have required emergency medical intervention and sutures after picking through a major blood vessel (such as the facial artery). One woman—who picked at a pimple on her neck with tweezers—lacerated her carotid artery, causing a near-fatal hemorrhage that required emergency surgery.⁸

 

Suicide risk. In a series of 123 patients with BDD, 33 (27%) excessively picked their skin and 10 of those who picked their skin (33%) had attempted suicide.5 In a case series of 31 patients with skin picking, 10% had attempted suicide.² We know of several young women whose chief complaint was skin picking and who committed suicide.⁵

Gender. The gender ratio of patients with skin picking remains unclear. In two case series that totaled 65 patients, 87% to 92% of those with pathologic skin picking were female.^2,7 In the series of patients with BDD, 58% of the 33 who compulsively picked their skin were female.⁵ On the other hand, most of 28 patients seen in a dermatology clinic for neurotic excoriations were male.⁹

Onset and chronicity. Pathologic skin picking may develop at any age, but it usually manifests in late adolescence or early adulthood, often after onset of a dermatologic illness such as acne² or in response to itching.³ Although long-term studies have not been done, the disorder appears to often be chronic, with waxing and waning of picking intensity and frequency.^1,2

Table 1

Skin picking: 3 steps to diagnosis and treatment

Step 1: Assess reasons for skin picking  Dermatologic or medical disorder? atopic dermatitis scabies Prader-Willi syndrome  Psychiatric disorder? body dysmorphic disorder obsessive-compulsive disorder delusional disorder dermatitis artefacta  Impulse control disorder, not otherwise specified?
Step 2: Assess picking severity  Treat comorbid mood or anxiety disorders  Treat skin picking if: patient is preoccupied with picking picking causes distress or dysfunction picking is causing skin lesions/disfigurement
Step 3: Provide recommended treatment  For adults  Habit reversal therapy plus medication is usually necessary  For children and adolescents  Habit reversal therapy alone for mild to moderate symptoms  Habit reversal therapy plus medication for severe symptoms

Comorbid psychopathology. In clinical settings, common comorbid psychopathologies include mood disorders (in 48% to 68% of patients with skin picking), anxiety disorders (41% to 64%), and alcohol use disorders (39%).²

In one patient sample, 71% of skin pickers met criteria for at least one personality disorder (48% had obsessive-compulsive personality disorder, and 26% met criteria for borderline personality disorder).²

Table 2

Medications with evidence of benefit for skin picking*

Medication	Dosage	Type of evidence
SSRIs
Citalopram	40 mg/d	Case report (effective only with inositol augmentation)16
Fluoxetine	20 to 80 mg/d	Case reports5,14-15 and two double-blind studies23-24
Fluvoxamine	100 to 300 mg/d	Case report,8 open-label study,21 and double-blind trial22
Sertraline	50 to 200 mg/d	Open-label study9
Other agents
Clomipramine	50 mg/d	Case report3
Doxepin	30 mg/d	Case report1
Naltrexone	50 mg/d	Case report20
Olanzapine	2.5 to 7.5 mg/d	Case report17
Pimozide	4 mg/d	Case report18
* Off-label uses; little scientific evidence supports using medications other than SSRIs for treating skin picking. Inform patients of the evidence for using any medication, risk of side effects including change in cardiac conduction (pimozide, clomipramine), seizure risk (pimozide, clomipramine), and tardive dyskinesia (pimozide), and potential interactions with other medications (all of the above).

PRIMARY VS. SECONDARY DISORDER

Is skin picking an independent disorder or a symptom of other psychiatric disorders? Although skin picking is not included in DSM-IV and has no formal diagnostic criteria, some forms of this behavior may belong among the impulse control disorders.

Patients often report an urge to pick their skin in response to increasing tension,^1,3 and picking results in transient relief or pleasure.^1,2 This description mirrors that of other impulse control disorders, such as trichotillomania and kleptomania. In fact, one study found that trichotillomania and kleptomania were common comorbidities among patients with skin picking (23% and 16%, respectively).² In 34 patients with psychogenic excoriation, only 7 (21%) appeared to have skin picking as a primary complaint, unaccounted for by another psychiatric disorder.⁷

Skin picking may also be a symptom of other psychiatric disorders. To determine whether another disorder is present, we ask patients why they pick their skin. Patients may be reluctant to reveal either the picking or the underlying disorder because of embarrassment and shame. The diagnosis can often be clarified by asking about the following conditions:

Body dysmorphic disorder. Nearly 30% of patients with BDD pick their skin to a pathologic extent.^5,6 The purpose of picking in BDD is to remove or minimize a nonexistent or slight imperfection in appearance (such as scars, pimples, bumps).^5,6

Obsessive-compulsive disorder. Patients with OCD may pick their skin in response to contamination obsessions.¹ Picking is often repetitive and ritualistic, and—as with compulsions—the behavior may reduce tension.¹⁰

Genetic disorders. Skin picking may be a symptom of Prader-Willi syndrome, a genetic disorder characterized by muscular hypotonia, short stature, characteristic facial features, intellectual disabilities, hypogonadism, hyperphagia, and an increased obesity risk. In one study, 97% of patients with Prader-Willi syndrome engaged in skin picking.¹¹

 

Delusional disorder. Delusions of parasitosis may result in skin picking, as patients attempt to remove imagined parasites or other vermin from on or under their skin.¹²

Dermatitis artefacta. Patients may consciously create skin lesions to assume the sick role. Onethird of patients presenting to dermatologists with a disease that is primarily psychiatric may be suffering from dermatitis artefacta.¹³

TREATMENT RECOMMENDATIONS

Successful clinical care of pathologic skin picking requires perseverance and patience from both patient and clinician.

Treatment begins with a thorough dermatologic examination for medical causes of skin picking (such as atopic dermatitis or scabies) and to treat excoriations (such as with antibiotics for infection). After the dermatologist has ruled out a medical cause, carefully assess the patient’s picking behavior and related psychiatric problems (Table 1).

• If picking is secondary to a psychiatric disorder, begin by providing appropriate treatment for that disorder.
• If picking results from BDD or OCD, we recommend habit reversal therapy combined with medication.
• If picking appears to be an independent impulse control disorder, simultaneous habit reversal therapy and medication is usually necessary to reduce symptoms.

SSRIs are a reasonable first medication because of evidence for their efficacy in reducing skin picking. Higher dosages—comparable to those used in treating OCD—are usually required to improve skin-picking behavior. You may need to try another SSRI if the first trial results in partial or no response.

In our experience, augmenting an SSRI with naltrexone, 50 mg/d, helps reduce intrusive urges to pick and is worth considering if SSRI therapy results in only partial response.

Children or adolescents. Depending upon symptom severity, a trial of habit reversal therapy may be appropriate before you recommend using medication.

EVIDENCE FOR DRUG THERAPY

Although few treatment studies have been done, skin picking does appear to respond to medication (Table 2).

Because no medications are approved to treat skin-picking behavior, inform patients of any “off-label” uses and the scientific or clinical evidence for considering medication treatment.

Case reports and case series. Selective serotonin reuptake inhibitors (SSRIs) appear most effective in patients with picking behavior, including:

• fluvoxamine, 300 mg/d, in one case report⁸
• fluoxetine, 20 to 80 mg/d, in several case reports.^5,14-15

In a series of 33 patients with BDD and compulsive skin picking, one-half (49%) of a variety of SSRI treatment trials improved BDD symptoms and skin picking behavior. The percentage of patients who improved was not examined. Dermatologic treatment alone was effective for only 15% of patients.⁵

Medications other than SSRIs have also been studied. One patient improved within 3 weeks of taking the tricyclic antidepressant clomipramine, 50 mg/d.³ Another patient picked her skin less often 4 weeks after inositol, 18 grams/d, was added to citalopram, 40 mg/d. Inositol, a nonprescription isomer of glucose, is a precursor in the phosphatidylinositol second-messenger cycle, which may play a role at certain serotonin receptors.¹⁶ The patient was given 6 grams dissolved in water three times daily.

Case reports have also suggested that olanzapine, pimozide, doxepin, and naltrexone may be beneficial in reducing skin excoriations. These reports often involved patients with psychiatric and medical comorbidities.^17-20

Table 3

Habit reversal: 5 components in patient learning

Awareness about picking behavior
Relaxation to reduce anxiety
Competing responses to learn behaviors incompatible with picking (such as fist clenching)
Rewarding oneself for successfully resisting picking
Generalizing the behavioral control

Open-label studies. In an open-label study of 28 patients with neurotic excoriation treated in a dermatology clinic, 68% improved within 1 month with sertraline, mean dosage 95 mg/d.⁹ Similarly, open-label fluvoxamine, mean dosage 112.5 mg/d, was effective in reducing skin excoriation in 7 of 14 patients treated for 12 weeks in a psychiatric setting.²¹

Double-blind studies. In a double-blind study using fluvoxamine with supportive psychotherapy in patients with psychocutaneous disorders, all five patients with acne excoriee improved after 4 weeks of medication treatment (none was randomized to placebo).²²

In a 10-week, double-blind study, 10 patients were assigned to fluoxetine, mean dosage 53.0 ± 16.4 mg/d, and 11 to placebo. A patient self-report visual analog scale showed that fluoxetine was significantly more effective than placebo in reducing picking behavior. Two other measures did not show significant improvement, however, perhaps because of the small sample size.²³

In a third study, 8 of 15 patients responded to open-label fluoxetine, 20 to 60 mg/d after 6 weeks. The responders were then randomized to 6 additional weeks of fluoxetine or placebo. All four patients assigned to continue active medication maintained their improvement. Symptoms returned to baseline by week 12 in the four assigned to placebo.²⁴

EVIDENCE FOR HABIT REVERSAL THERAPY

No controlled trials have examined psychosocial treatments for skin picking, but several psychotherapeutic interventions appear promising. Habit reversal has shown promise in three case reports totaling seven patients and appears to reduce picking behavior within a few weeks.^25-27

 

In a case series, three patients were successfully treated with habit reversal (Table 3) and cognitive-behavioral techniques, consisting of:

• awareness training (using a skin-picking diary)
• competing response techniques (such as making a fist or squeezing a ball)
• emotion regulation skills
• psychoeducation
• cognitive restructuring (such as using Socratic questioning to produce rational alternatives) in situations that elicit the urge to pick.²⁸

In another case series, 22 dermatology patients with skin picking received psychotherapy with insight-oriented and behavioral components. Therapy included attention to developmental issues and active conflicts, cognitive restructuring, and tools to manage aggression and social relations. Although treatment duration varied— the mean was weekly for 14 months—skin lesions healed in 17 patients (77%).²⁹

Related resources

• Obsessive-Compulsive Foundation http://www.ocfoundation.org
• Koran LM. Obsessive-compulsive and related disorders in adults: A comprehensive clinical guide. Cambridge, UK: Cambridge University Press, 1999.
• Phillips KA. The broken mirror: Recognizing and treating body dysmorphic disorder. New York: Oxford University Press, 1996.

Drug brand names

• Citalopram • Celexa
• Clomipramine • Anafranil
• Doxepin • Sinequan
• Fluoxetine • Prozac
• Fluvoxamine • Luvox
• Naltrexone • ReVia
• Olanzapine • Zyprexa
• Pimozide • Orap
• Sertraline • Zoloft

Disclosure

Dr. Grant reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.

Dr. Phillips receives research support from Eli Lilly and Co., Forest Pharmaceuticals, and Gate Pharmaceuticals; she is a speaker for or consultant to Eli Lilly and Co., Forest Pharmaceuticals, and UCB Pharma.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.

What is kleptomania? An independent illness, a symptom of other psychiatric disorders, or merely criminal behavior? Kleptomania—a disorder defined by an inability to resist the impulse to steal—is one of psychiatry’s most poorly understood diagnoses, even though it has been recognized in the literature for almost 200 years.

Kleptomania causes notable distress and impaired functioning.¹ People with kleptomania often suffer from comorbid mood, anxiety, substance use, and other impulse-control disorders.¹⁴ They experience the humiliation of repeated arrests, which leads to guilt, depression, and even suicide.^1,5 Yet kleptomania usually goes undiagnosed and untreated, despite a lifetime prevalence as high as 0.6%.⁶

Case report: ‘I’m a thief’

“I’m a thief,” began Susan, age 39. “I steal something four or five times every week. I steal from grocery stores and clothing stores. Sometimes I might steal something like vanilla extract; other times an expensive men’s tie. I probably steal $200 worth of items every week.

“You probably won’t believe this, but I don’t want or need the stuff I take. I have plenty of money. I have no idea why I take the things I do. That’s why I’m so depressed. What kind of person does something like this?

The urge to steal “I was probably 14 when I started stealing. I would go to stores with my mother. When I saw certain objects, I would get urges to steal them. The odd thing was that the items I stole were so ridiculous. I remember stealing key chains for several months, maybe three or four times a week. When I got older, things got worse. I was having urges more often, and so I needed to steal more often.

Box

“My entire life has been torment. Each day I worry about having the urges, and then I worry about being caught stealing. I can’t relax. I’ve been married for 17 years, and I haven’t told my husband. My secrecy is tearing our marriage apart. My husband thinks I’m having an affair because I’ve distanced myself emotionally from him.”

Table 1

SCREENING TEST FOR KLEPTOMANIA

	Yes	No
1. Do you steal or have urges to steal?	○	○
2. Do thoughts of stealing or urges to steal preoccupy you? That is, do you often think about stealing or have urges to steal and wish the thoughts or urges occurred less often?	○	○
3. Do you feel tense or anxious before you steal or when you have urges to steal?	○	○
4. Do you feel pleasure or a sense of calm when you steal something?	○	○
5. Has the stealing or urges to steal caused you much distress?	○	○
6. Has the stealing or urges to steal significantly interfered with your life in some way?	○	○
A patient who answers “yes” to questions 1 through 4 and to question 5 or 6 is likely to have kleptomania.
Adapted from DSM-IV criteria, American Psychiatric Association, 2000

Susan described urges to steal almost every day. When the urges were mild, she could resist them. Other days they were severe, and Susan felt unable to control her behavior. At work, her urges distracted her from completing projects, and her performance suffered. The urge to steal would often compel Susan to leave work early so she could get to a store.

Calm, then guilt “Every time I steal something I feel both a thrill and a great sense of calm,” she said. “It feels good. The problem is that almost immediately after each theft, I feel guilty and ashamed. After I steal, I usually donate the items to the Salvation Army, throw them away, or give them away as gifts.”

Drug trials We started treating Susan with the selective serotonin reuptake inhibitor (SSRI), citalopram. She reported notable improvement in her mood after 3 weeks on a dosage of 60 mg/d and she had been attending weekly psychotherapy, although her stealing continued unchanged. The addition of naltrexone, 200 mg/d for 2 weeks, decreased the frequency of Susan’s stealing and reduced her urges to steal, but her symptoms continued to interfere significantly with her overall functioning.

We then added the atypical antipsychotic quetiapine, 100 mg bid, and Susan’s urges to steal and stealing behavior went into remission within 3 weeks. She has refrained from stealing for the last 9 months.

 

Making the diagnosis

In our clinic, we have treated more than 50 patients with kleptomania. Rather than coming to us through the criminal justice system, they are usually self-referred. Often they contact us after discovering on the Internet that we specialize in treating persons with this disorder.

In our experience, kleptomania typically goes undiagnosed in clinical settings, in part because patients are ashamed and embarrassed to discuss their symptoms with physicians unless specifically asked.¹ If left untreated, however, kleptomania frequently becomes chronic.⁴ If persons with kleptomania are to seek treatment, it is important that family, friends, and mental health professionals understand the myths and facts about this disorder (Box).

To make the diagnosis, we use the simple screening instrument shown in Table 1.⁷ In general, because of high comorbidity with certain disorders, we screen every patient presenting to our clinic with a mood, substance use, anxiety, or eating disorder, or who has a problem with impulse control. Kleptomania is likely if the patient answers “yes” to questions 1 through 4 and to question 5 or 6. Stealing may be a symptom of several other psychiatric disorders, however, and misdiagnosis is fairly common (Table 2).^6-8

Data suggest that the female-to-male ratio in kleptomania is approximately 2:1, with onset in adolescence. Typical individuals with kleptomania steal because they have urges to steal, often triggered by specific stimuli such as the sights and sounds of stores or feelings of loneliness or stress.¹ Most patients with kleptomania are fairly specific about the types of stores from which they steal and the items they steal, and most hoard stolen items.¹

Although many patients with kleptomania function quite well, others are severely debilitated in social and occupational realms. In a series of 22 patients with kleptomania:

• 64% had been apprehended
• 23% had served jail time
• 27% had been hospitalized because of their kleptomania symptoms
• 18% had considered or attempted suicide because of the distress associated with their kleptomania.¹

Treatment recommendations

Patient history With patients who steal, we begin by identifying the motivation behind the stealing. Most patients with kleptomania report urges to steal. Some of these patients may have comorbid depression; for them, stealing makes them feel less depressed. Anger or irritability may point to borderline personality disorder. Stealing for the enjoyment of risk may suggest bipolar disorder.

Table 2

DIFFERENTIAL DIAGNOSIS: DISORDERS THAT MAY INVOLVE STEALING

Misdiagnosis	How to distinguish from kleptomania
Bipolar disorder	Patients with bipolar disorder may steal as a result of the impulsivity of mania. In fact, the diagnostic criteria for kleptomania require the exclusion of mania as the cause of stealing.7 Patients with bipolar disorder report an elevated, expansive, or irritable mood while stealing. Patients with kleptomania tend to report a depressed mood when not stealing
Borderline personality disorder	Unlike patients with borderline personality disorder, patients with kleptomania do not report long histories of unstable relationships or negative self-image; inappropriate anger and “psychotic-like” symptoms are rare in patients with kleptomania
Antisocial personality disorder (ASPD, or conduct disorder)	Patients with kleptomania suffer intense shame and guilt, unlike those with ASPD. Also, most patients with kleptomania do not report other illegal or antisocial behavior.
Eating disorders	Data suggests that about one-third of patients with an eating disorder also steal.6,8 Patients with kleptomania, however, do not have disordered eating patterns or distorted body images common to patients with eating disorders.

Many patients with kleptomania have comorbid mood, substance, or anxiety disorders. Treating these other symptoms while ignoring the symptoms of kleptomania may be unsuccessful. Comorbidity also may influence the choice of medication.

Medical assessment Case reports have associated the onset of kleptomania with a variety of medical conditions, including presenile cortical atrophy in a 25-year-old, a parietal tumor that caused blackouts and obliterated any memory of stealing episodes, narcolepsy, and an insulinoma that caused severe hypoglycemia.⁹ The relationship of these conditions with the onset of kleptomania is unclear, but the reports suggest that medical causes—although unlikely—should be ruled out before you consider kleptomania as a psychiatric illness.

Patient education Persons with kleptomania often feel that no one else has the same problem. They do not think of their behavior as being an illness. It is helpful to explain that kleptomania is treatable and to connect patients with educational books, self-help groups, and Web sites providing information and support (see “Related resources”).

Cognitive-behavioral therapy (CBT) Although the evidence is quite limited, covert sensitization, exposure and response prevention, and imaginal desensitization have all been shown effective in case reports.¹⁰

What medications are effective?

Only case reports, a case series of five subjects, and a single open-label treatment study involving 10 subjects with kleptomania have been done.

 

So far, uses of tricyclic antidepressants (imipramine, nortriptyline), SSRIs (fluoxetine, fluvoxamine, paroxetine), the opioid antagonist naltrexone, and mood stabilizers (lithium, valproate) have met with varying degrees of success. Strategies targeting urge and behavior reduction and mechanisms for coping with urges and behavior (e.g., cognitive-behavioral therapies) may represent important adjunctive components.^2,11-17

No medications are FDA-approved for treating kleptomania. Therefore, it is important to inform patients of any off-label use of medications for this disorder, as well as the empirical basis for considering pharmacologic treatment.

SSRIs Only case reports exist on the use of SSRIs in treating kleptomania. The disorder may share a common pathology with pathologic gambling, and in our clinical experience appears to respond to similar treatments.¹⁸ We draw on research of pathologic gambling as well as our clinical experience in choosing SSRIs as first-line treatment, especially for patients with significant mood symptoms.¹⁹

We suggest titrating SSRIs to the maximum recommended dosage. As in the treatment of pathologic gambling, dosages of SSRIs required to treat kleptomania symptoms appear to be higher than average dosages required to treat depressive disorders. An SSRI should not be considered ineffective unless it has been tried for at least 10 to 12 weeks and the highest dosage tolerated or recommended by the manufacturer has been reached.

Response to SSRIs usually is characterized by decreased thoughts about stealing, decreased stealing behavior, and improvement in social and occupational functioning. If an SSRI is only partially effective, we consider augmentation with naltrexone, buspirone, or a mood stabilizer.

Naltrexone Patients taking naltrexone often report less-intense urges to steal. The urges may not disappear but are often sufficiently reduced so that the patient can resist them more easily. Patients also report that the thrill associated with stealing is reduced or eliminated.

Naltrexone was used in the first medication study of kleptomania and showed a significant decline in the intensity of urges to steal, stealing thoughts, and stealing behavior. Average dosage was 150 mg/d;¹¹ a reduced dosage (e.g., 50 mg/d) may work in adolescents with kleptomania.²⁰

Nausea as a side effect can be reduced by starting patients on 25 mg/d for the first 3 or 4 days and possibly adding ondansetron, 4 to 8 mg/d. Nausea and diarrhea are usually mild and resolve within the first week. Clinically, most patients respond to naltrexone within 2 weeks. After that, the dosage usually needs to be adjusted.

In patients with comorbid depression, augmentation with an SSRI may prevent worsening of untreated depressive symptoms. It is prudent to obtain liver function tests prior to naltrexone administration and again 3 to 4 weeks after starting the drug.²¹ Repeat testing should be performed at 2-to 4-week intervals for the next 2 months, then once a month for the following 3 months. After 6 months, testing three to four times a year is usually sufficient.

Nonsteroidal analgesics should not be used with high dosages of naltrexone (>50 mg/d), as concurrent use may increase the risk of hepatic transaminase elevation.²¹

Mood stabilizers Responses to lithium and valproate have been described in two case reports of patients with kleptomania.^14,15 In the case of valproate, the effective dosage was 2,000 mg/d, whereas lithium reduced stealing urges at a serum level of 0.5 mEq/L.

Although it would be premature to recommend the use of mood stabilizers, their possible benefit may be related to their efficacy in bipolar disorder treatment and the existence of features (e.g., impulsivity) shared by kleptomania and bipolar disorder.

Atypical antipsychotics Although there is no evidence that atypical antipsychotics are useful in kleptomania, augmenting an SSRI with an atypical neuroleptic may be beneficial. Atypical antipsychotics have been explored as augmenting agents in the treatment of nonpsychotic disorders and behaviors, including pathologic gambling and obsessive-compulsive disorder.

The role of psychotherapy

Cognitive-behavioral therapy Based on the evidence of its effectiveness in treating pathologic gambling, CBT may hold promise as monotherapy for mild cases of kleptomania.

Combination therapy Combined pharmacologic and behavioral therapy may be the optimal treatment strategy for kleptomania. In our experience, patients who respond only partially or fail to respond to pharmacotherapy alone are more likely to find relief with a combination of drug and cognitive-behavioral therapies.

Related resource

• Goldman MJ. Kleptomania: the compulsion to steal—what can be done? Far Hills, N New Horizon Press, 1998.
• Shoplifters Alternative http://www.shoplifters.org
• Impulse Control Disorders Clinic, University of Minnesota http://www.med.umn.edu/psychiatry/research/impulse.htm

Drug brand names

• Citalopram • Celexa
• Fluvoxamine • Luvox
• Imipramine • Tofranil
• Naltrexone • Revia
• Nortriptyline • Aventyl, Pamelor
• Paroxetine • Paxil
• Quetiapine • Seroquel
• Valproic acid • Depakote

Disclosure

The authors report no affiliation or financial arrangement with any of the companies whose products are mentioned in this article.