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Imaging Study Offers New Insight on RA's Pathology

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RA May Not Actually Start in the Synovium

In this study, the MRI findings showed that bone edema was a significant precursor for development of erosions. Ultrasound measures synovitis that is secondary to inflammation, but it is unable to depict inflammation in the subchondral bone. For many years, we thought that the origin of rheumatoid arthritis was primarily in the synovium and that it progressed from the synovium into the joint or back into the subchondral bone. As the authors of the accompanying editorial point out, it would appear from this analysis that there is a disconnect between the inflammation and the synovium and the subchondral bone. This notion has been written about before (Arthritis Rheum. 2007;56:1118–24). What this disconnect shows us is that there are inflammatory changes occurring in the subchondral bone as evidenced by bone marrow edema. The findings suggest that RA may actually start in either the synovium or in the marrow of the subchondral bone. Alternatively, it may occur simultaneously in the subchondral bone and the synovium. Thus, although ultrasound is a very strong measurement of synovitis, it does not tell us what is going on in the subchondral bone, which is a very important area in the RA overview. Once RA patients begin treatment with biologics, MRI provides crucial information on treatment response by assessing any changes in bone marrow edema from baseline. Although oncologists take it for granted that imaging has a role in assessing treatment response in patients with cancer or lymphoma, we are not yet comfortable with that concept in RA. As is reported, MRI allows us to see synovial and bone marrow changes including osteitis, whereas ultrasound is limited to measuring synovial changes and erosions. Because RA may have two components to it – the synovium and the subchondral bone – the ideal imaging tool is probably the MRI or a CT scan, but the CT is not a practical tool because it exposes patients to excessive radiation.

Norman B. Gaylis, M.D., is president of the International Society of Extremity MRI in Rheumatology and president of Arthritis and Rheumatic Disease Specialties in Aventura, Fla. He has no relevant financial conflicts to disclose.


 

Erosive progression is arrested in rheumatoid arthritis patients who are treated with adalimumab and methotrexate combination therapy, judging from results of a novel longitudinal study comparing MRI, ultrasonography, CT, and radiography.

However, only MRI was sensitive enough to document repair of individual erosions. Both MRI and ultrasound could detect changes in bone edema, which "was predictive of subsequent erosive progression on CT, both at the individual bone/joint level and also for MRI bone edema at the patient level," reported lead author Dr. Uffe Møller Døhn of the department of rheumatology at the University of Copenhagen in the February 2011 edition of the Annals of the Rheumatic Diseases. "These data emphasize the predictive value of modern imaging, and especially highlight the importance of MRI bone edema for predicting erosive progression."

The findings also offer a different way of thinking about RA’s pathology. In an accompanying editorial, Dr. Fiona McQueen and Dr. Esperanza Naredo said that the study’s findings add to existing evidence suggesting that osteitis is more strongly predictive of bone erosion than is synovitis, which supports "the notion that there is a more direct connection between bone inflammation and bone damage than between synovial inflammation and bone damage" (Ann. Rheum. Dis. 2011;70:241-4). They described synovitis and osteitis as "cousins with a common ancestor, the process that ultimately drives both remaining obscure but possibly sited in the bone marrow."

Dr. Døhn and his associates used MRI, ultrasound, standard radiography, and high-resolution CT images of the wrist and metacarpophalangeal joints 2-5 to study response to adalimumab/methotrexate therapy in 52 RA patients naive to biological agents. All images were obtained before the first dose of adalimumab injection and were repeated after 6 and 12 months of treatment (Ann. Rheum. Dis. 2011;70:252-8).

The median age of patients was 61 years, and 67% were women. From baseline, the researchers did not observe any statistically significant changes in overall bone destruction or repair at 6 or 12 months, but differences were seen when researchers used the smallest-detectable-change cutoff. For example, after 6 and 12 months, the scores of MRI synovitis, grey-scale synovitis, and power Doppler ultrasonography decreased. So did scores as assessed by DAS28 (disease activity score in 28 joints), a health assessment questionnaire, and tender and swollen joint counts.

Study participants with disease progression on CT had higher baseline MRI bone edema scores. In fact, when baseline MRI bone edema was present, the risk ratio for erosive progression in the same bone on CT at 12 months was 3.8. In addition, time-integrated MRI bone edema, power Doppler, and grey-scale synovitis scores were higher in bones and joints with CT progression.

With CT as the reference method, the researchers determined that the sensitivity and specificity for the other imaging modalities were 68% and 92%, respectively, on MRI; 44% and 95% on ultrasonography; and 26% and 98% on radiography.

In their concluding remarks in the editorial, Dr. McQueen and Dr. Naredo emphasized that the reduction of both synovitis and osteitis "is clearly an important therapeutic goal" in treating RA. "The detection and monitoring of synovitis is often more feasible in clinical practice using [ultrasound] than MRI scanning, but the latter does afford the opportunity to detect and monitor bone edema at the same time."

Dr. McQueen is with the department of molecular medicine and pathology at the University of Auckland (New Zealand). Dr. Naredo is with the department of rheumatology at the Hospital Universitario Severo Ochoa in Madrid.

Funding for the study was provided by Abbott Denmark, the Danish Rheumatism Association, and the Aase and Ejner Danielsen Foundation. Dr. Døhn reported no relevant financial conflicts, but many of the study’s coauthors disclosed that they have received consulting fees, speaking fees, or research grants from Abbott, Centocor, Roche, Schering-Plough, UCB-Nordic, and Wyeth. Dr. McQueen and Dr. Naredo stated that they have no conflicts of interest.

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